SENS6 (Strategies for Engineered Neglible Senescene) Conference has began 3th September 2013 in Queen's College Cambridge,UK. So,the delegate of the young russian students in the head of Alex Zhavoronkov (Biogerontology Research Foundation) are also participate in this action where Anastasia Shindyapina will have a 15 minutes talk session (September 6th) in order to her project "Mineralization of the connective tissue: a complex molecular process leading to age-related loss-of-function" and Alex Zhavoronkov will represent us with an important part of the age-related issues such as " New economic arguments for accelerating aging research" ( September 6th).
At the first session " Calorie restriction in primates" the foreign scientists (Ingram and Anderson) represented a complex research associated with the calorically restricted diet affect the lifespan extension and aging-related diseases in the different organisms ranged from unicellular to non-human primates by presenting summaries of more than 20 years of longitudial data gathered at the their institutios.
So, Donald Ingram ( Pennington Biomedical Research Center,USA ) showed in his work "Is calorie a calorie"that after 150 rhesus monkyes studies including monkeys with CR (Calorie Restriction) and a control group, the CR group are healtheir that fully-food counterparts based on reduced incidence of chronic diseases,exhobot significally better indices of predisposition to disease.
Rozalyn Anderson (University of Winfileston) in her work " impact of CR on aging in rhesus monkeys-a focus on metabolism". So,analysis of metabolic rate and energy indicate that the CR animals differ from controls in metabolic efficiency. Also, in CR animals the onset of sarcopenia is delayed and multiple indicators of cellular metabolism just prove a "younger" profile.
In the end of the session Luigi Fontana ( Washington University,USA/Sakerano University,Italy) discussed about the implications of the primate data for calorie restriction in human. The difference between the 2 above stidies is caused by various diet composition,hormonal and metabolic adaptions and so on. However, we don't currenty know about the CR possibillities in human lifespan extension.
At the second session "Small -molecule interventions effective at late age" Dongheng Cai (Albert Einstein College of Medicine,USA) reported about "Hypotalamic control of ging and longevity".So,it is known that IKK/NF-kB activation affects the adult neural stem cells fate leading to aging and metabolic syndrome conditions.By the way, Cai and his scientific group found that NF-kB signalling in the hypothalamus can positively and negatively regulate multiple aging phenotypes in mice and the mediation of this effect in the restof the body by gonadotropin- relesing hormone.
Danica Chen (Univesity of California at Berkley,USA),"Sirtuin regulation of metabilism and stem cells" supposed that SIRT 3 reactivation can induce rejuvination in older organisms.
Frank Modeo (Unoversity of Graz,Austria),"Spermidine induces autopology in multiple species and mimics calorie restriction" found that external administration of spemidine troggers aitopothology and prolongs lifespan of yeasts,flies and worms in authophagy dependent manner.
During the sessions all delegates can discuss the interest and exciting questions with a cup of cofee and delicious breakfast/lunch/dinner meetings.
At the first session " Calorie restriction in primates" the foreign scientists (Ingram and Anderson) represented a complex research associated with the calorically restricted diet affect the lifespan extension and aging-related diseases in the different organisms ranged from unicellular to non-human primates by presenting summaries of more than 20 years of longitudial data gathered at the their institutios.
So, Donald Ingram ( Pennington Biomedical Research Center,USA ) showed in his work "Is calorie a calorie"that after 150 rhesus monkyes studies including monkeys with CR (Calorie Restriction) and a control group, the CR group are healtheir that fully-food counterparts based on reduced incidence of chronic diseases,exhobot significally better indices of predisposition to disease.
Rozalyn Anderson (University of Winfileston) in her work " impact of CR on aging in rhesus monkeys-a focus on metabolism". So,analysis of metabolic rate and energy indicate that the CR animals differ from controls in metabolic efficiency. Also, in CR animals the onset of sarcopenia is delayed and multiple indicators of cellular metabolism just prove a "younger" profile.
In the end of the session Luigi Fontana ( Washington University,USA/Sakerano University,Italy) discussed about the implications of the primate data for calorie restriction in human. The difference between the 2 above stidies is caused by various diet composition,hormonal and metabolic adaptions and so on. However, we don't currenty know about the CR possibillities in human lifespan extension.
At the second session "Small -molecule interventions effective at late age" Dongheng Cai (Albert Einstein College of Medicine,USA) reported about "Hypotalamic control of ging and longevity".So,it is known that IKK/NF-kB activation affects the adult neural stem cells fate leading to aging and metabolic syndrome conditions.By the way, Cai and his scientific group found that NF-kB signalling in the hypothalamus can positively and negatively regulate multiple aging phenotypes in mice and the mediation of this effect in the restof the body by gonadotropin- relesing hormone.
Danica Chen (Univesity of California at Berkley,USA),"Sirtuin regulation of metabilism and stem cells" supposed that SIRT 3 reactivation can induce rejuvination in older organisms.
Frank Modeo (Unoversity of Graz,Austria),"Spermidine induces autopology in multiple species and mimics calorie restriction" found that external administration of spemidine troggers aitopothology and prolongs lifespan of yeasts,flies and worms in authophagy dependent manner.
During the sessions all delegates can discuss the interest and exciting questions with a cup of cofee and delicious breakfast/lunch/dinner meetings.
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